Presenter(s)
D. Sabater Minarim1, A. Dornisch2, R. Karunamuni3, K. M. Morgan4, T. J. Nelson5, C. Teerlink6, J. A. Lynch7, I. Garraway8, V. Million Veteran Prog9, T. M. Seibert3, J. Vassy10, and B. S. Rose2; 1Department of Radiation Medicine and Applied Sciences, UC San Diego Health, San Diego, CA, 2Department of Radiation Medicine and Applied Sciences, University of California San Diego, La Jolla, CA, 3Research Service, VA San Diego Healthcare System, San Diego, CA, 4Center for Health Equity, Education and Research, University of California San Diego, La Jolla, CA, 5VA San Diego Health Care System, La Jolla, CA, 6Veteran Affairs Medical Center, Salt Lake City, UT, 7Department of Internal Medicine, University of Utah School of Medicine, Salt Lake City, UT, 8VA Greater Los Angeles Health System, Los Angeles, CA, 9U.S. Department of Veterans Affairs, Washington, DC, 10VA Boston Healthcare System, Boston, MA, USA, Boston, MA
Results: Black/AA men had higher mean PHS601 z-scores compared to NHW men (0.94 vs. -0.27, p<0.001). In the clinical variables only model, Black/AA race was associated with significantly higher odds of cs-PC (OR=1.73, 95% CI: 1.61-1.85, p<0.001). After adding both polygenic scores, the race effect was substantially attenuated (OR=1.07, 95% CI: 0.99–1.16, p=0.099). PHS601 was associated with cs-PC (OR=1.75, 95% CI: 1.69-1.82, p<0.001), while PRS447 was inversely associated (OR=0.69, 95% CI: 0.67-0.72, p<0.001). PHS601 mediated ~90% of this racial disparity (Average Causal Mediation Effect=0.127, 95% CI: 0.119-0.135, p<0.001). The residual direct effect was non-significant (Average Direct Effect=0.014, 95% CI: -0.001-0.031, p=0.072).
Conclusion: The higher rate of cs-PC diagnosis among Black/AA veterans is largely explained by elevated risk as captured by PHS601. These findings support the use of genetically informed screening strategies, replacing self-reported race as a screening determinant with a more precise and interpretable measure of individual risk.